Case 1:

JF is a 42-year old previously healthy farmer who presented with sudden onset central chest pain of one hour duration to the ED. The pain is associated with sweating and radiates to the left arm and jaw. The area of discomfort is about the size of the fist.

Ragavendra R. Baliga, M.D
Assistant Professor
Division of Cardiology
University Of Michigan

 

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Question 3 of 39:

On examination his heart rate is 90 beats per minute, blood pressure is 140/100 mm Hg, he has normal heart sounds and his lungs are clear to auscultation.

This EKG shows that this patient has an inferior wall myocardial infarction.

You clicked 'False'

Correct!

The initial abnormality during a transmural myocardial infarction is elevation of ST segment, often with a peaked appearance of the T wave. After the first few minutes of an infarction the T waves become tall, pointed and upright and there is ST elevation. Typically ECG changes are usually confined to the leads that 'face' the infarction. Therefore, an inferior wall myocardial infarction is diagnosed when ECG findings are seen in leads II, III and aVF. Lateral infarction produces changes in leads I, aVL and V5/6. Reciprocal changes (ST depression) may be seen in leads V1 and V2 in inferior wall MI. Because no leads are placed on the patients back overlying the posterior wall, the clinician relies on chest leads V1 and V2 which are directly opposite the posterior wall and these record the inverse of what leads on the back would record. Therefore, ST depression will be seen in these leads.

Infarct Site

Leads with abnormal EKG changes

Coronary Artery Most Often Implicated

Anterior

Small

Extensive

V3-V4

V2-V5

V1-V6

LAD

Anteroseptal

V1-V3

LAD

Septal

V1-V2

LAD

Anterolateral

V4-V6, aVL

CFX

Lateral

I, aVL

CFX

Apical

V5-V6

CFX

Inferior

II, III aVF

RCA

Posterior

V1-V2 (reciprocal)

RCA

Subendocardial

Any lead

 

 

 
 
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