Bin HU, M.D., Ph.D.

Research Investigator

Education: M.D., Binzhou Medical College, Binzhou, People's Republic of China; Ph.D. (Immunology), Shinshu University School of Medicine, Matsumoto, Japan Clinical Training: Pediatric Hematology, Graduate School in Tianjin Medical College, Tianjin, People's Republic of China Research Training:  Postdoctoral research fellowship, Pulmonary Immunology, Pulmonary & Critical Care Medicine Division, University of Michigan (Curtis Lab)  Major Research Interests: Molecular mechanisms controlling macrophage recognition and ingestion of apoptotic cells Care Unit Monitoring and Outcomes

ROM and We

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· Yasui K, Hu B, Nakazawa T, Agematsu K, Komiyama A.  Theophylline accelerated human granulocyte apoptosis not via phosphodiesterase inhibition. J Clin Invest100:1677-84, 1997.

· Hu B, Sonstein J, Christensen PJ, Punturieri A, Curtis JL.  Deficient in vitro and in vivo phagocytosis of apoptotic T cells by resident murine alveolar macrophages.  J Immunol 165:2124-2133, 2000. 

· Hu B, Punturieri A, Todt J, Sonstein J, Polak T, Curtis JL.  Recognition and phagocytosis of apoptotic T cells by resident murine tissue macro-phages requires multiple signal transduction events.  J Leukoc Biol 71; 881-889, 2002.

· Canetti C, Hu B, Curtis JL, Peters-Golden M.  Syk activation is a leukotriene B4-regulated event involved in macrophage phagocytosis of IgG-coated targets but not apoptotic cells.  Blood 102; 1877-1883, 2003. 

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